| acvr1b | |||||||||||||||||||||||||||||
| Type | Gene1 | Effect | Gene2 | Sentence | Perturbation methods | Time of Interaction in evidence | territories of interaction or Tissue | Evidence for Direct | Authors | Journal | PubYear | Volume | Pages | MedLine Reference | Connectivity | # of References | Found In Pathways | Journal Link | Journal Reference | Mechanism | URN | ISSN | Issue | LocatorString | MedlineTA | PubMonth | p-value [Evol. Bioinformatics 2007:3(197)] | TextPath | Title |
| Binding | acvr1b | neutral | acvr2a | If cotransfected with ActRII/IIB and ALK4, Cripto inhibited crosslinking of activin to ALK4 and the association of ALK4 with ActRII/IIB. | J. Biol. Chem | 12682303:6 | 2 | 13 | Zebrafish_FoxH1_20080728 | http://www.jbc.org/cgi/content/full/270/11/6308 | v270 i11 p6308 | urn:agi-Binding:inout-urn:agi-llid:92:inout-urn:agi-llid:91: | digtext:http://www.sciencedirect.com/science?_ob=GatewayURL&_origin=inwardhub&_urlversion=4&_method=citationSearch&_piikey=S1534580701000764&_referrer=www.ncbi.nlm.nih.gov/pubmed/11709181?ordinalpos=10&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsP... | ||||||||||||||||
| Expression | acvr1b | positive | ntl | Injection of TARAM-A* (acvr1b) induced very robust expression of ntl in large patches of cells. | overexpression | 4.33 | margin | 2 | Thierry Lepage | Development | 2002 | 129 | 4901 | ||||||||||||||||
| Expression | acvr1b | positive | og9x | Injection of TARAM-A*(acvr1b) induced very robust expression of casanova (sox32) in large patches of cells. | overexpression | 5.25 | margin | 2 | Thierry Lepage | Development | 2002 | 129 | 4901 | ||||||||||||||||
| Expression | acvr1b | positive | pitx2a | We report here that similar to effects of ectopic TGF betas, right-side expression of constitutively active activin-like kinase (ALK) 4 results in LR organ reversals as well as altered pitx2a expression in the lateral plate mesoderm. | mesoderm | 15063168:2 | 2 | 3 | Zebrafish_smad2_20080729 | urn:agi-Expression:inout-urn:agi-llid:91:out-urn:agi-llid:5308::positive | digtext:http://www.ncbi.nlm.nih.gov/entrez/utils/fref.fcgi?PrId=3048&itool=AbstractPlus-def&uid=16684517&db=pubmed&url=http://linkinghub.elsevier.com/retrieve/pii/S0012-1606(06)00257-0 | ||||||||||||||||||
| ProtModification | acvr1b | phosphorylation | smad | The binding of Nodal to its receptor complex (Alk4/ActRIIB/co-receptor EGF-CFC) activates Smad2/3, and phosphorylation of these Smads by Alk4 increases their affinity for Smad4. | 2 | 1 | Zebrafish_smad2_20080729 | phosphorylation | urn:agi-ProtModification:inout-urn:agi-llid:91:out-urn:agi-go:0005075::unknown | digtext:http://www.ncbi.nlm.nih.gov/entrez/utils/fref.fcgi?PrId=3051&itool=AbstractPlus-def&uid=18039968&db=pubmed&url=http://dev.biologists.org/cgi/pmidlookup?view=long&pmid=18039968 | |||||||||||||||||||
| Binding | acvr1b | neutral | smad | The ?4-?5 loop of TaramA, a Smad interacting domain, is identical to that of Alk4 ( ten Dijke et al., 1994b), suggesting that TaramA and Alk4 may interact with similar downstream Smad molecules and may perform similar functions.When CA alk8 and taramA* RN | 2 | 2 | Zebrafish_Bmp2b_20080727 | urn:agi-Binding:inout-urn:agi-llid:91:inout-urn:agi-go:0005075: | digtext:http://www.ncbi.nlm.nih.gov/entrez/utils/fref.fcgi?PrId=3048&itool=AbstractPlus-def&uid=11165484&db=pubmed&url=http://linkinghub.elsevier.com/retrieve/pii/S0925477300005414 | ||||||||||||||||||||
| ProtModification | acvr1b | phosphorylation | smad2 | This suggests that Smad2 and Smad3 association with ALK4 is an early event after receptor heteromerization, in activin signaling. | egg | Mol. Endocrin | 9892009:1092 | 2 | 5 | acvr2a B-acvr1b | http://mend.endojournals.org/cgi/content/full/13/1/15 | v13 i1 p15 (1999) | phosphorylation | urn:agi-ProtModification:inout-urn:agi-llid:91:out-urn:agi-llid:4087::unknown | digtext:C:\Documents and Settings\yuh\Desktop\zebrafish GRN Literatures\Gerster-T-1999.pdf | url(C%3a%5cDocuments%20and%20Settings%5cyuh%5cDesktop%5czebrafish%20GRN%20Literatures%5cGerster-T-1999.pdf):pdf | |||||||||||||
| Expression | acvr1b | positive | smad2 | Ca-ALK4 induced a strong nuclear accumulation of EGFP-Smad2 during the 20 min washout period, and this was completely abolished by the treatment with AA (Figure 4A). | 2 | 1 | Zebrafish_smad2_20080729 | urn:agi-Expression:inout-urn:agi-llid:91:out-urn:agi-llid:4087::positive | digtext:http://www.sciencedirect.com/science?_ob=GatewayURL&_origin=inwardhub&_urlversion=4&_method=citationSearch&_piikey=S1534580707000111&_referrer=www.ncbi.nlm.nih.gov/pubmed/17276343?ordinalpos=7&itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPa... | ||||||||||||||||||||
| ProtModification | acvr1b | phosphorylation | smad3a | ALK4 is a Ser/Thr kinase that phosphorylates cytoplasmic Smad2 and Smad3 ( Massague and W otton, 2000 ). | 2 | 6 | Zebrafish GRN from literatures-7-24-2008 | phosphorylation | urn:agi-ProtModification:inout-urn:agi-llid:91:out-urn:agi-llid:4088::unknown | digtext:C:\Documents and Settings\yuh\Desktop\zebrafish GRN Literatures\Dougan-ST-2007b.pdf | url(C%3a%5cDocuments%20and%20Settings%5cyuh%5cDesktop%5czebrafish%20GRN%20Literatures%5cDougan-ST-2007b.pdf):pdf | ||||||||||||||||||
| Binding | acvr1b | neutral | smad3a | Nevertheless, when cells were stimulated by activin, the amount of Smad3 associated with ALK4 increases. | Mol. Endocrin | 9892009:1073 | 2 | 3 | Zebrafish_smad2_20080729 | http://mend.endojournals.org/cgi/content/full/13/1/15 | v13 i1 p15 (1999) | urn:agi-Binding:inout-urn:agi-llid:91:inout-urn:agi-llid:4088: | digtext:http://www.ncbi.nlm.nih.gov/entrez/utils/fref.fcgi?PrId=3051&itool=AbstractPlus-def&uid=18039968&db=pubmed&url=http://dev.biologists.org/cgi/pmidlookup?view=long&pmid=18039968 | ||||||||||||||||
| Expression | acvr1b | positive | sox17 | Injection of TARAM-A* induced very robust expression of sox17, ntl,og9x, bon/mixer and casanova in large patches of cells. | overexpression | 4.33 | margin | 2 | Thierry Lepage | Development | 2002 | 129 | 4901 | ||||||||||||||||
| Expression | acvr1b | positive | sox17 | Similarly, whereas injection of very low doses 1-2 pg of tar*/acvr1b caused the ectopic expression of sox17 in about 32% of the embryos, treatment with SU5402 following the injection increased this percentage to 54% (Table 1). | 2 | 9 | Zebrafish GRN from literatures-7-24-2008 | urn:agi-Expression:inout-urn:agi-llid:91:out-urn:agi-llid:64321::positive | digtext:C:\Documents and Settings\yuh\Desktop\zebrafish GRN Literatures\Lepage-T-2006.pdf | url(C%3a%5cDocuments%20and%20Settings%5cyuh%5cDesktop%5czebrafish%20GRN%20Literatures%5cLepage-T-2006.pdf):pdf | |||||||||||||||||||
| Expression | acvr1b | positive | sox32 | Embryo injected at the eight-cell stage into one blastomere with 40 pg of TARAM-A*(acvr1b) and with 100 pg of mRNA encoding a nuclear b-Gal (blue colour) as a lineage tracer. TARAM-A*(acvr1b) induces confluent patches of mezzo (og9x)-expressing cells. | overexpression | 4.33 | margin | 2 | Thierry Lepage | Development | 2002 | 129 | 4901 | ||||||||||||||||
| Regulation | acvr1b | unknown | tgfb1 | This phenomenon was also observed in MZoep mutant embryos treated with the small compound SB431542 (39), which specifically inhibits ALK4/5/7-mediated TGF-? signaling. | 2 | 1 | Zebrafish_smad2_20080729 | urn:agi-Regulation:inout-urn:agi-llid:91:out-urn:agi-llid:7040::unknown | digtext:http://www.ncbi.nlm.nih.gov/entrez/utils/fref.fcgi?PrId=3051&itool=AbstractPlus-def&uid=18025082&db=pubmed&url=http://www.jbc.org/cgi/pmidlookup?view=long&pmid=18025082 | ||||||||||||||||||||
| Binding | acvr1b | neutral | tgfb1 | Together, these results suggest not only that ALK4 interacts with multiple TGF?s to generate embryonic pattern, but also that ALK4 ligands differentially utilize the ALK4 pathway to regulate distinct aspects of axial pattern, with Vg1 as a modulator of AL | 2 | 1 | Zebrafish_smad2_20080729 | urn:agi-Binding:inout-urn:agi-llid:91:inout-urn:agi-llid:7040: | digtext:http://www.ncbi.nlm.nih.gov/entrez/utils/fref.fcgi?PrId=3048&itool=AbstractPlus-def&uid=16684517&db=pubmed&url=http://linkinghub.elsevier.com/retrieve/pii/S0012-1606(06)00257-0 | ||||||||||||||||||||
| ProtModification | acvr1b | phosphorylation | ttrap | ALK4 phosphorylated TTRAP in vitro (Fig. 6A). | 2 | 1 | Zebrafish_smad2_20080729 | phosphorylation | urn:agi-ProtModification:inout-urn:agi-llid:91:out-urn:agi-llid:51567::unknown | digtext:http://www.ncbi.nlm.nih.gov/entrez/utils/fref.fcgi?PrId=3051&itool=AbstractPlus-def&uid=18039968&db=pubmed&url=http://dev.biologists.org/cgi/pmidlookup?view=long&pmid=18039968 | |||||||||||||||||||
| Binding | acvr1b | neutral | ttrap | Weobserved binding of Alk4 to TTRAP (Fig. 5D). | 2 | 1 | Zebrafish_smad2_20080729 | urn:agi-Binding:inout-urn:agi-llid:91:inout-urn:agi-llid:51567: | digtext:http://www.ncbi.nlm.nih.gov/entrez/utils/fref.fcgi?PrId=3051&itool=AbstractPlus-def&uid=18039968&db=pubmed&url=http://dev.biologists.org/cgi/pmidlookup?view=long&pmid=18039968 | ||||||||||||||||||||
| Expression | bmp2a | negative | acvr1b | overexpression of BMPs compromises endoderm formation, formation of endoderm precursors is negatively regulated by BMPs on the ventral side. | overexpression | 5.25 | blastoderm | 2 | Thierry Lepage | Development | 2006 | 133 | 2189 | ||||||||||||||||
| Expression | bmp2b | negative | acvr1b | We never found a homozygous laftm110b embryo that had a wild-type or ventralized phenotype, consistent with the Laf/Alk8 receptor functioning downstream of the Bmp2b. | overexpression | 5.25 | blastoderm | 2 | Thierry Lepage | Development | 2006 | 133 | 2189 | ||||||||||||||||
| Expression | bmp7 | negative | acvr1b | Prdm1 expression is reduced in snh (BMP7) mutant embryos. | overexpression | 5.25 | blastoderm | 2 | Thierry Lepage | Development | 2006 | 133 | 2189 | ||||||||||||||||
| Expression | fgf17 | negative | acvr1b | activation of the FGF/ERK pathway disrupts endoderm formation in the embryo and antagonizes the ability of an activated form of Tar/Acvr1b to induce endoderm at the animal pole. | overexpression | 5.25 | blastoderm | 2 | Thierry Lepage | Development | 2006 | 133 | 2189 | ||||||||||||||||
| Expression | fgf24 | negative | acvr1b | Microinjection of fgf24 leads to inhibition of bmp2b, bmp4 and bmp7 expression at blastula stage. | overexpression | 5.25 | blastoderm | 2 | Thierry Lepage | Development | 2006 | 133 | 2189 | ||||||||||||||||
| Expression | fgf3 | negative | acvr1b | Microinjection of fgf3 leads to inhibition of bmp2b expression at blastula stage. | overexpression | 5.25 | blastoderm | 2 | Thierry Lepage | Development | 2006 | 133 | 2189 | ||||||||||||||||
| Expression | fgf8 | negative | acvr1b | The fgf8 overexpression (10 pg) induced chd at the shield stage. | overexpression | 5.25 | blastoderm | 2 | Thierry Lepage | Development | 2006 | 133 | 2189 | ||||||||||||||||
| Regulation | mapk1 | negative | acvr1b | FGF/ERK activity antagonizes the ability of tar*/acvr1b to induce endoderm. | 2 | 8 | Zebrafish GRN from literatures-7-24-2008 | urn:agi-Regulation:inout-urn:agi-llid:5594:out-urn:agi-llid:91::negative | digtext:C:\Documents and Settings\yuh\Desktop\zebrafish GRN Literatures\Lepage-T-2006.pdf | url(C%3a%5cDocuments%20and%20Settings%5cyuh%5cDesktop%5czebrafish%20GRN%20Literatures%5cLepage-T-2006.pdf):pdf | |||||||||||||||||||